Annual Review of Biochemistry· 2017Q1· Review
Protein Misfolding, Amyloid Formation, and Human Disease: A Summary of Progress Over the Last Decade
- 2,704citations
- Q1SCImago
- 2017year
Short summary
Proteins can misfold into amyloid aggregates, linked to diseases like Alzheimer's, Parkinson's, and type II diabetes, with recent advances clarifying fibril structures, formation mechanisms, and the role of proteostasis networks.
AI-generated from the title and abstract; the full text is not read.
TakeawaysIn the app
Key pointsIn the app
Ask the paperIn the app
The rest is in the Pofolia app
Takeaways, key points and questions to the paper; new summaries every day for your field. Free.
Sign in on the web to openField: Physiology
PhysiologyMedicine