Neurology (Neuroscience) papers

Pofolia’s corpus holds 74 papers from Neurology (Neuroscience) (2012–2026), each with a short summary. Below, the 20 most-cited, then the most recently added.

At a glance

• Most cited: Copper-Zinc Superoxide Dismutase (SOD1) Is Released by Microglial Cells and Confers Neuroprotection against 6-OHDA Neurotoxicity (2012, 9,017 citations).
• The 20 papers listed have 61,627 citations between them.
• 17 of the 19 with a known journal quartile appeared in a Q1 journal.
• 15 have a free full text (open access).
• Most frequent journals: Clinical Neurophysiology, New England Journal of Medicine, Science.
• Published between 2012 and 2020.

Most cited

Ranked by citation count. Because citations accumulate over time, this list naturally leans towards work published a few years ago; for where the field is now, see “recently added”. How to read the signals

• Copper-Zinc Superoxide Dismutase (SOD1) Is Released by Microglial Cells and Confers Neuroprotection against 6-OHDA Neurotoxicity

  Neurosignals · 2012 · Q4 · FWCI 193.36 · 9,017 citations · Open access

  Microglia release copper-zinc superoxide dismutase (SOD1), which protects neurons from 6-hydroxydopamine (6-OHDA) induced toxicity by increasing calcium influx.

  Go to source →

• Changes of Intestinal Functions in Liver Cirrhosis

  Inflammatory Intestinal Diseases · 2016 · Q3 · FWCI 358.52 · 6,814 citations · Open access

  Liver cirrhosis significantly alters intestinal functions, including dysmotility, prolonged transit times, and bacterial overgrowth (SIBO), which are linked to disease severity and complications like hepatic encephalopathy.

  Go to source →

• The Neurology of Eye Movements

  Oxford University Press eBooks · 2015 · FWCI 102.70 · 3,805 citations

  This new edition offers a modern synthesis of the neural basis for eye movements, integrating anatomical, physiological, and pharmacological data with current understanding of reflexive and voluntary gaze control.

  Go to source →

• Complement and microglia mediate early synapse loss in Alzheimer mouse models

  Science · 2016 · Q1 · SJR 10.00 · FWCI 130.20 · 3,392 citations

  In Alzheimer's disease models, complement protein C1q and microglia drive early synapse loss before plaque formation, a process previously thought to occur later and be driven by neuroinflammation.

  Go to source →

• The Blood–Brain Barrier

  Cold Spring Harbor Perspectives in Biology · 2015 · Q1 · SJR 3.00 · 3,301 citations · Open access

  The blood-brain barrier (BBB) is a unique system of blood vessels in the central nervous system that tightly regulates the passage of substances into the brain, crucial for neuronal function and protection.

  Go to source →

• TREM2 Variants in Alzheimer's Disease

  New England Journal of Medicine · 2012 · Q1 · SJR 18.00 · FWCI 93.49 · 3,090 citations · Open access

  Rare heterozygous variants in the TREM2 gene significantly increase the risk of developing Alzheimer's disease, with a specific variant (R47H) showing a strong association.

  Go to source →

• Microglia Function in the Central Nervous System During Health and Neurodegeneration

  Annual Review of Immunology · 2017 · Q1 · SJR 16.00 · FWCI 58.16 · 2,844 citations · Open access

  Microglia, the brain's resident immune cells, are crucial for development, neuronal maintenance, and repair, but their altered function is implicated in neurodegeneration.

  Go to source →

• Non-invasive electrical and magnetic stimulation of the brain, spinal cord, roots and peripheral nerves: Basic principles and procedures for routine clinical and research application. An updated report from an I.F.C.N. Committee

  Clinical Neurophysiology · 2015 · Q1 · SJR 1.00 · FWCI 95.93 · 2,786 citations

  This report updates previous guidelines on non-invasive electrical and magnetic stimulation techniques for the brain, spinal cord, and nerves, reflecting advancements since 1994.

  Go to source →

• Variant of TREM2 Associated with the Risk of Alzheimer's Disease

  New England Journal of Medicine · 2012 · Q1 · SJR 18.00 · FWCI 84.52 · 2,629 citations

  A rare mutation in the TREM2 gene (rs75932628-T, R47H substitution) significantly increases the risk of late-onset Alzheimer's disease by nearly threefold.

  Go to source →

• Neuroinflammation in neurodegenerative disorders: the roles of microglia and astrocytes

  Translational Neurodegeneration · 2020 · Q1 · SJR 4.00 · FWCI 77.85 · 2,551 citations · Open access

  This review highlights that microglia and astrocytes, key regulators of neuroinflammation in diseases like Alzheimer's and Parkinson's, exhibit complex and heterogeneous phenotypes beyond a simple neurotoxic vs. neuroprotective dichotomy.

  Go to source →

• Genomic Analysis of Reactive Astrogliosis

  Journal of Neuroscience · 2012 · Q1 · SJR 1.00 · FWCI 47.04 · 2,476 citations · Open access

  Reactive astrocytes, crucial for CNS injury response, exhibit distinct molecular phenotypes depending on the specific injury type, with at least 50% of gene expression changes being injury-specific.

  Go to source →

• Evidence-based guidelines on the therapeutic use of repetitive transcranial magnetic stimulation (rTMS): An update (2014–2018)

  Clinical Neurophysiology · 2020 · Q1 · SJR 1.00 · FWCI 91.20 · 2,317 citations · Open access

  Updated guidelines confirm definite efficacy (Level A) for high-frequency rTMS of the motor cortex for neuropathic pain and left prefrontal cortex for depression, and low-frequency rTMS of the motor cortex for stroke motor recovery.

  Go to source →

• Global, regional, and national burden of neurological disorders during 1990–2015: a systematic analysis for the Global Burden of Disease Study 2015

  The Lancet Neurology · 2017 · Q1 · SJR 12.00 · FWCI 96.69 · 2,300 citations · Open access

  Neurological disorders are the leading cause group of disability globally, accounting for 10.2% of all DALYs in 2015, and the second-leading cause of death. Despite falling age-standardized rates, the absolute number of deaths from these disorders rose 36.7% between 1990 and 2015.

  Go to source →

• Inflammation as a central mechanism in Alzheimer's disease

  Alzheimer s & Dementia Translational Research & Clinical Interventions · 2018 · Q1 · SJR 1.00 · FWCI 60.23 · 2,269 citations · Open access

  Inflammation, driven by sustained activation of brain immune cells like microglia, is now recognized as a third core pathology in Alzheimer's disease (AD), exacerbating amyloid and tau pathologies.

  Go to source →

• Blood-Brain Barrier: From Physiology to Disease and Back

  Physiological Reviews · 2018 · Q1 · SJR 11.00 · FWCI 58.23 · 2,184 citations · Open access

  This review synthesizes current understanding of the blood-brain barrier (BBB), detailing its physiological functions, mechanisms of breakdown in neurological diseases, and potential therapeutic applications.

  Go to source →

• Microglial M1/M2 polarization and metabolic states

  British Journal of Pharmacology · 2015 · Q1 · SJR 2.00 · FWCI 37.87 · 2,074 citations · Open access

  This review explores how microglia, the brain's immune cells, shift between inflammatory (M1) and repair (M2) states, and how their energy metabolism, particularly mitochondrial activity, changes accordingly.

  Go to source →

• Microglia in Alzheimer’s disease

  The Journal of Cell Biology · 2017 · Q1 · SJR 3.00 · FWCI 40.65 · 1,981 citations · Open access

  Microglia, immune cells in the brain, play a dual role in Alzheimer's disease (AD), potentially offering protection while also contributing to neuronal damage and pathology.

  Go to source →

• How neuroinflammation contributes to neurodegeneration

  Science · 2016 · Q1 · SJR 10.00 · FWCI 82.08 · 1,975 citations

  Neuroinflammation, driven by changes in the brain's supportive glial cells and blood-brain barrier, is a key factor in the progression of neurodegenerative diseases like Alzheimer's and Parkinson's.

  Go to source →

• Mechanisms regulating intestinal barrier integrity and its pathological implications

  Experimental & Molecular Medicine · 2018 · Q1 · SJR 5.00 · FWCI 37.82 · 1,922 citations · Open access

  The intestinal epithelial cell layer, supported by tight junction proteins, acts as a crucial physical barrier against external factors. Imbalances in its regulation are linked to inflammatory bowel disease, obesity, and metabolic disorders.

  Go to source →

• New tools for studying microglia in the mouse and human CNS

  Proceedings of the National Academy of Sciences · 2016 · Q1 · SJR 3.00 · FWCI 85.88 · 1,900 citations · Open access

  Researchers have identified Transmembrane Protein 119 (Tmem119) as a highly specific marker for microglia in both mouse and human brains, enabling new tools for their study.

  Go to source →

Recently added

• Correlation analysis between cerebrovascular intervention therapy and perioperative blood pressure variability and cerebral blood flow reserve in elderly patients with intracranial artery stenosis

  BMC Neurology · 2026 · Q2

  Elderly patients with intracranial artery stenosis undergoing intervention therapy face a significantly higher risk of poor prognosis if they experience high perioperative blood pressure variability (BPV) and a decreased rate of cerebral blood flow velocity (CBFV), with a combined effect increasing risk by 8.77 times.

  Go to source →

• sTREM2 and GFAP mediated the association of cerebrospinal fluid GLP-1R with tau pathology in alzheimer’s disease

  Aging Clinical and Experimental Research · 2026 · Q1 · SJR 1.00

  Lower cerebrospinal fluid (CSF) GLP-1R levels are linked to increased tau pathology in Alzheimer's disease, with sTREM2 and GFAP acting as partial mediators.

  Go to source →

• Thalamic projections from the cerebellar nuclei receiving proprioceptive signals from the jaw muscles in rats

  Brain Structure and Function · 2026 · Q1 · SJR 1.00

  Rats' jaw muscle proprioceptive signals project from cerebellar nuclei (jcms-IntDL, jcms-MedDL) to distinct thalamic regions, with jcms-IntDL targeting VL, VM, ventral anterior, PC, and OPC, and jcms-MedDL targeting parafascicular, centrolateral, VL, VM, and PC nuclei.

  Go to source →

• Coenzyme A activates TLR4 and enhances alternative macrophage activation in line with MyD88 agonism

  EMBO Reports · 2026 · Q1 · SJR 2.00

  Exogenous coenzyme A (CoA) acts as a weak TLR4 agonist, priming macrophages for enhanced IL-4 signaling and alternative activation, leading to inflammation resolution.

  Go to source →

• A framework and practical guide for conducting double-blind randomized clinical trials combining transcranial electrical stimulation and psychotherapy

  Translational Psychiatry · 2026 · Q1 · SJR 2.00

  A new framework and practical guide addresses methodological challenges for designing double-blind randomized clinical trials that combine transcranial electrical stimulation (tES) with psychotherapy.

  Go to source →

• Neuromodulatory mechanism of repetitive transcranial magnetic stimulation for freezing of gait in Parkinson’s disease

  BMC Medical Imaging · 2026 · Q1

  Repetitive transcranial magnetic stimulation (rTMS) targeting the primary motor cortex (M1) improved freezing of gait (FOG) in Parkinson's disease (PD) patients, evidenced by enhanced gait performance and restoration of aberrant brain activity, including increased cortical inhibition and improved white matter integrity.

  Go to source →

• Exploring key genes associated with metabolic cell death in ischemic stroke: a comprehensive analysis of single-cell, bulk transcriptome, and experimental validation

  Scientific Reports · 2026 · Q1 · Open access

  Five genes (Atf3, Cdkn1a, Hmox1, Jun, Ptgs2) involved in metabolic cell death were identified as key players in ischemic stroke (IS), with a nomogram model of these genes showing good diagnostic performance for IS incidence.

  Go to source →

• Limitation of pantothenate bioavailability in Anopheles stephensi by second-generation pantazines enhances restriction of Plasmodium development

  Scientific Reports · 2026 · Q1

  Second-generation pantazines (PZ-3022 and PZ-3883) significantly reduced pantothenate levels in Anopheles stephensi mosquitoes, leading to a marked decrease in Plasmodium oocyst and sporozoite burdens.

  Go to source →

Journals in this field

The journals that publish most of this field’s papers. Quartile (Q1–Q4), SJR and h-index are from SCImago Journal Rank; “in this field” is how many of the journal’s pooled papers belong here. What is a Q1 journal? · What is the h-index?

Every journal in Neurology (Neuroscience) (Q1–Q4) →

Other subfields in Neuroscience

• Cognitive Neuroscience
• Biological Psychiatry
• Behavioral Neuroscience
• Sensory Systems
• Endocrine and Autonomic Systems
• Developmental Neuroscience
• Cellular and Molecular Neuroscience

All subfields of Neuroscience →

← All fields